Introduction
Coronavirus (COVID-19) infection exposes patients with heart failure to
a higher risk of morbidity and mortality.[1] Asymptomatic or mildly
symptomatic COVID-19 respiratory virus infections can include fever,
coughing, tiredness, dyspnea, diarrhea, headaches, and myalgia.
Respiratory rate >30 bpm, arterial oxygen saturation less
than 93% at rest, development of acute respiratory distress syndrome,
septic shock, metabolic acidosis, and coagulopathy, including
disseminated intravascular coagulation, are characteristics of severe
cases. [2] In addition, patients infected with the COVID-19 virus
have an increased risk of developing both venous and arterial thrombosis
[3-6]
Thrombocytopenia and an increase in D-dimer are the hemostatic changes
caused by COVID-19 that are most frequently observed, and they are
linked to a greater need for mechanical ventilation, admission to
intensive care, and death. It has been noticed that patients who are
older and have comorbid conditions are more likely to have a high
mortality rate, [7] which is believed to be greatly influenced by
blood vessel dysfunction and clot formation, as indicated by elevated
D-dimer levels brought on by blood clots.[8] Despite the that VAD
thrombosis is a common complication, LVAD thrombosis due to COVID -19 is
underreported and few cases are presented in the literature. Here we
present a case of accelerated LVAD outflow thrombosis in the setting of
COVID-19 infection with multiorgan failure.